![]() RésuméĪu cours d’un exercice de forte intensité, la phosphocréatine (PCr) intracellulaire se scinde rapidement pour favoriser le renouvellement de l’adénosine triphosphate (ATP), ce qui explique la large utilisation du monohydrate de créatine en tant que facteur ergogène. Although the mechanism for this is not fully understood, it may be related to the asymmetrical distribution of muscle mass around those joints. The results suggest that short-term supplementation with creatine monohydrate reduces the active ROM of shoulder extension and abduction and of ankle dorsiflexion. There was also a significant increase in body mass for the CS group (83.6 ± 6.2 kg vs. Data indicated significant reductions in active ROM in 3 movements: shoulder extension (57 ☑1.3° PRE vs. Before (24 h before starting supplementation (PRE) and after (on the 8th day of supplementation (POST)) this loading phase, both groups underwent goniometry measurement of the shoulder, elbow, hip, and ankle.
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